INTEGRATIVE OMICS IN PARKINSON'S DISEASE.

Integrative Omics in Parkinson's Disease provides a comprehensive understanding of the current literature on high-throughput technologies relating to discoveries for Parkinson's disease etiology.This emerging field uses large omics datasets to investigate the etiology of Parkinson's d...

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Bibliographic Details
Corporate Author: ScienceDirect (Online service)
Other Authors: Trinh, Joanne
Format: eBook
Language:English
Published: [S.l.] : Academic Press, [2025].
Subjects:
Online Access:Connect to the full text of this electronic book
Table of Contents:
  • Future directions and conclusions
  • Acknowledgments
  • References
  • Chapter 5: Methods to investigate somatic structural variants in synucleinopathies
  • Introduction
  • Combined immunofluorescence-FISH for the detection of somatic SNCA CNVs
  • Single-cell whole genome sequencing
  • Digital PCR and droplet digital PCR for the detection of low-level SNCA CNVs
  • Long-read sequencing: Targeted and genome-wide
  • Future directions
  • Acknowledgment
  • References
  • Chapter 6: Mitochondrial genetics in Parkinsons disease
  • Abbreviations
  • Introduction
  • Mitochondria in Parkinsons disease
  • The mitochondrial genome-Organization and different forms of variations
  • Mitochondrial DNA copy number
  • Copy number variations in Parkinsons disease
  • Methods to assess mitochondrial copy number
  • Optical and biochemical mtDNA-CN estimation
  • PCR-based mtDNA-CN estimation
  • Microarray-based mtDNA-CN estimation
  • Next-generation sequencing-based mtDNA-CN estimation
  • Structural variants of the mitochondrial genome
  • mtDNA structural variation in PD
  • 7S DNA in PD
  • mtDNA deletions in PD
  • Methods to assess structural variation of mitochondrial genomes
  • PCR-based methods
  • NGS-based methods
  • Long-read sequencing
  • Germline variants and mitochondrial haplogroups
  • Association of mtDNA single nucleotide variants or haplogroups with PD
  • Assessment of mitochondrial germline variants
  • Array-based genotyping
  • Sequencing
  • mtDNA variant annotation and pathogenicity scores
  • Mitochondrial heteroplasmy
  • Heteroplasmic variant load is related to Parkinsons disease
  • Different approaches to assess heteroplasmy
  • Understudied mitochondrial DNA variations
  • Epigenetics
  • Cell-free mitochondrial DNA
  • Conclusion
  • References
  • Chapter 7: DNA methylation studies in Parkinsons disease
  • The most studied epigenetic mechanism in complex diseases.
  • The new era of transcriptomics: Long-read RNA sequencing
  • Applications of long-read sequencing
  • Elucidating gene function
  • Variant interpretation
  • Long-read RNA sequencing can aid in gene therapy design
  • Long-read transcriptomics in neurodegenerative disease
  • MAPT in Alzheimers disease
  • SNCA in Parkinsons disease
  • GBA1 and GBAP1 in Parkinsons disease
  • TARDBP in amyotrophic lateral sclerosis and frontotemporal dementia
  • The future of long-read sequencing in neurotranscriptomics
  • Challenges in using long-read RNA sequencing when studying neurodegenerative diseases
  • Concluding remarks
  • References
  • Chapter 11: Gene-environment interactions and behavior
  • Historical background
  • Metaanalyses and systematic reviews investigating environmental factors and PD risk
  • Age at onset in PD
  • Models to study gene-environment interactions
  • Systematic review of gene-environment interactions in the literature
  • Conclusion and outlook
  • References
  • Chapter 12: Introduction to prediction modeling using machine learning and omics data
  • Introduction
  • Machine learning approaches for prediction modeling
  • Training a prediction model
  • Measuring prediction performance
  • Validating a prediction model
  • Explaining a prediction model
  • Further reading
  • Acknowledgment
  • References
  • Chapter 13: Merging iPSCs and ``omics´´: Advances in the field and potential applications to untangle neurodegenerative d ...
  • Introduction
  • iPSCs and neurons
  • Neurodegenerative diseases
  • Parkinsons disease
  • Alzheimers disease
  • Amyotrophic lateral sclerosis
  • Disease mechanisms identified in iPSC-based studies using omics
  • Merging iPSCs and ``omics´´
  • Multiomics approach to untangle neurodegenerative diseases
  • Parkinsons disease
  • Alzheimers disease
  • Amyotrophic lateral sclerosis
  • Conclusions and limitations.
  • Translational potential and future perspectives
  • References
  • Index.