Cellular, molecular, and environmental contribution in cardiac remodeling : from lab bench work to its clinical perspective /

Cellular, Molecular and Environmental Contribution in Cardiac Remodeling: From Lab Bench to Clinical Perspective consolidates the most recent research advances on cellular, molecular, biochemical, and heterogeneous factors contributing to the physiological and pathological cardiac remodeling, elucid...

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Bibliographic Details
Main Author: Duttaroy, Asim K.
Corporate Author: ScienceDirect (Online service)
Other Authors: Mallick, Rahul
Format: eBook
Language:English
Published: London : Academic Press, 2024.
Subjects:
Online Access:Connect to the full text of this electronic book
Table of Contents:
  • Front Cover
  • Cellular, Molecular, and Environmental Contribution in Cardiac Remodeling
  • Cellular, Molecular, and Environmental Contribution in Cardiac Remodeling: From Lab Bench Work to Its Clinical Perspective
  • Copyright
  • Contents
  • Preface
  • 1
  • Cardiac remodeling: Impacts on cardiac health and disease
  • 1. Introduction
  • 2. Physiological cardiac remodeling
  • 3. Exercise-induced cardiac remodeling
  • 4. Pregnancy-induced cardiac remodeling
  • 5. Pathological cardiac remodeling
  • 6. Hypertensive left ventricular remodeling
  • 7. Atrophic cardiac remodeling
  • 8. Ventricular remodeling in myocardial infarction
  • 9. Metabolic remodeling of the heart
  • 10. Diagnosis
  • 11. Consequences
  • 12. Congestive heart failure
  • 13. Therapeutic targets
  • 14. Fibrosis
  • 15. Cardiomyocyte hypertrophy
  • 16. Cardiomyocyte death
  • 17. Vascular remodeling
  • 18. Metabolic remodeling
  • 19. Conclusion
  • References
  • 2
  • Physiological cardiac modeling: Effects of exercise and other physical activities
  • 1. Introduction
  • 2. Effects of exercise and cardiovascular system
  • 3. Effects of exercise on the cardiac remodeling
  • 4. Effects of exercise on the left and right ventricles
  • 5. Determinants of exercise-induced cardiac remodeling magnitude
  • 6. Cellular and molecular mechanism of mechanisms of EICR
  • 7. Energy metabolism, exercise, and cardiac remodeling
  • 8. Cellular signaling system, exercise, and cardiac remodeling
  • 9. Conclusions
  • References
  • 3
  • Prognostic elements of unfavorable cardiac remodeling
  • 1. Introduction
  • 2. Left ventricular ejection fraction
  • 3. Myocardial infarction
  • 4. Ventricular dilatation
  • 5. Wall thickness
  • 6. Biomarkers
  • 7. Hypertrophy
  • 8. Fibrosis
  • 9. Hemodynamic changes
  • 10. Functional impairment
  • 11. Symptom severity
  • 12. Comorbidities
  • 13. Response to treatment.
  • 14. Summary
  • References
  • 4
  • Neurohormones in cardiac remodeling and function
  • 1. Introduction
  • 2. Neurohormonal activation
  • 3. The adrenergic nervous system in the heart
  • 3.1 Adrenergic nervous system regulates cardiac inflammation
  • 3.2 Adrenergic nervous system regulation in heart failure
  • 3.3 Beta-adrenergic dysfunction on aging heart
  • 3.4 Comparison between heart failure and aging heart
  • 3.5 How safe to target the adrenergic nervous system to treat heart failure
  • 4. RAAS perturbation
  • 5. RAAS in cardiac remodeling
  • 6. Conclusion
  • References
  • 5
  • Cardiokines and cardiac remodeling
  • 1. Introduction
  • 2. The heart as an endocrine organ
  • 3. Cardiokines in cardiac stress
  • 4. Autocrine/paracrine signaling of cardiokines
  • 5. Endocrine activities of cardiokines
  • 6. Cardiokines as cardiac disorders biomarkers
  • 7. Conclusion
  • References
  • 6
  • Comparative effects of fatty acid and glucose in cardiac remodeling
  • 1. Introduction
  • 2. Cardiac hypertrophy impacting fatty acids and glucose metabolism
  • 3. Glucose and cardiac remodeling
  • 4. Fats and cardiac remodeling
  • 5. Conclusions
  • References
  • 7
  • Vitamins, minerals, and nutraceuticals: Their cardioprotective functions
  • 1. Introduction
  • 2. Cardiac protection by vitamins
  • 3. Minerals and cardiac remodeling
  • 4. Phenolic compounds and cardiac remodeling
  • 5. Conclusions
  • References
  • 8
  • Junctional adhesion molecules: Their roles in integrity and functionality of the heart
  • 1. Introduction
  • 2. Interactions and signaling pathways
  • 3. Major functions
  • 4. Role of junctional adhesion molecules in cardiovascular diseases
  • 5. Conclusion and perspective
  • References
  • 9
  • Composition and function of ion channels and their effects on cardiac remodeling
  • 1. Introduction.
  • 4. Regulation of myosin
  • 5. Actin-myosin structural transition in heart diseases
  • 6. Cardiac troponin: Biomarker of cardiac damage
  • 7. Conclusion
  • References
  • 15
  • Growth and proliferation of cardiomyocytes: Roles of energy metabolism, cell death, oxidative stress, and meta ...
  • 1. Introduction
  • 2. Microanatomy
  • 3. Development of the heart
  • 4. Cardiomyocyte cell cycle and regeneration potential
  • 5. Cardiomyocyte senescence in adult heart
  • 6. Cross-talk between cardiomyocytes and cardiac resident other cells during cardiac remodeling
  • 7. Clinical perspective
  • 8. Conclusion
  • References
  • 16
  • Cardiac endothelial cells and their cross-talks with neighboring cells in cardiac remodeling
  • 1. Introduction
  • 2. Cardiac endothelial cell transcriptome
  • 3. Functional significance of endothelial cells during cardiac remodeling
  • 3.1 Mechanosensing and mechanoregulation
  • 4. Maintenance of vascular tone
  • 5. Hemostasis
  • 6. Role in neovascularization
  • 7. Act as conditional immune cells
  • 8. Interaction with cardiomyocytes
  • 9. Cross-talk with leukocytes
  • 10. Differentiation into mesenchymal cells
  • 11. Contribute to cardiac fibrosis
  • 12. Recruitment of endothelial progenitor cells
  • 13. Conclusion
  • References
  • 17
  • Pathophysiology of cardiac fibroblasts and impacts on the severity of the cardiac disease
  • 1. Cardiac fibroblasts
  • 2. Cardiac-resident fibroblast activation
  • 3. Origins and characterization of cardiac fibroblasts
  • 4. Fibroblast responses to microenvironmental stimuli
  • 5. Prime regulator of extracellular matrix turnover
  • 6. Electrophysiology of cardiac fibroblasts
  • 7. Contribution to cardiac fibrosis
  • 8. Conclusion
  • References
  • 18
  • Involvement of cardiac stem cells in cardiac remodeling or myocardial regeneration
  • 1. Introduction
  • 2. Self-renewal cells in the heart.
  • 3. Embryological origin of cardiac stem cells
  • 4. Endogenous cardiac stem and progenitor cells
  • 5. The regenerative capacity of cardiac stem and progenitor cells
  • 6. Metabolism of cardiac stem cells
  • 7. Paracrine role of cardiac stem cells following cardiac injury
  • 8. Cross-talk between cardiac stem cells and other cells
  • 9. Clinical prospect
  • References
  • 19
  • Cardiac pericytes and cardiac remodeling
  • 1. Introduction
  • 2. Properties of cardiac pericytes
  • 3. Pericyte secretome
  • 4. Contribution to cardiac homeostasis
  • 5. Contribution to the cardiac remodeling process
  • 6. Are cardiac pericytes and cardiac stem cells similar
  • 7. Therapeutic potential of cardiac pericytes
  • 8. Conclusion
  • References
  • 20
  • Macrophages in the remodeling of diseased heart
  • 1. Introduction
  • 2. Identification of cardiac macrophages
  • 3. Origin of cardiac macrophages
  • 4. Role of macrophages during cardiac remodeling
  • 5. Mechanism of macrophage polarization
  • 6. Role of macrophages in ventricular arrhythmia
  • 7. Conclusion
  • References
  • 21
  • Inflammatory role of neutrophils in cardiac remodeling: Damage versus resolution
  • 1. Introduction
  • 2. Phenotypic heterogeneity of neutrophils
  • 3. Contribution in adaptive immunity
  • 4. Does circadian oscillation affect the neutrophil counts: Prognostic factor of disease outcome?
  • 5. Dual role of neutrophil infiltration kinetics in cardiac remodeling: Damage versus resolution?
  • 6. NETosis
  • 7. Secreted extracellular vesicle-mediated effects in the heart
  • 8. Provoking granulopoiesis during myocardial infarction
  • 9. Production of specialized proresolving mediators
  • 10. Angiogenic properties
  • 11. Sexual dimorphism in postmyocardial infarction inflammatory responses
  • 12. Conclusion
  • References
  • 22
  • Extracellular vesicle in cardiac remodeling
  • 1. Introduction.