DNA alterations in cancer : genetic and epigenetic changes /

Bibliographic Details
Other Authors: Ehrlich, Melanie, 1945-
Format: Book
Language:English
Published: Natick, MA : Eaton Pub., [2000]
Subjects:
Table of Contents:
  • Sect. 1. Introduction
  • Introduction to cancer genes and growth control / Nesrin Özören, Wafik S. El-Deiry
  • Sect. 2. Tumor suppressor genes and cancer modifier loci
  • Rb and other pocket proteins, chromatin remodeling, and cancer / Slimane Ait-Si-Ali ... [et al.]
  • The hedgehog/patched signaling pathway: the role of tumor suppressor genes in development / Michael Dean
  • Structure-function relationship of the INK4 family of tumor suppressors / Junan Li ... [et al.]
  • The role of genetic modifiers, both mutations and polymorphisms, and environmental factors in cancer susceptibility / Bharati Bapat, Susmita Esufali
  • Sect. 3. Activation of the oncogenic function of proto-oncogenes and other cancer promoting genes by mutation or rearrangement
  • Multiple roles for deregulated MYC expression in oncogenesis / Amato J. Giaccia, Nicholas C. Denko
  • Misregulation of a signal transduction pathway: the role of Rel/NF-B transcription factors in oncogenesis / Thomas D. Gilmore, Jean-Charles Epinat, Margaret Barkett
  • Fusion oncoproteins resulting from chromosomal translocations and leukemogenesis / Randy Fenrick, Scott W. Hiebert
  • Dissecting the genetics of the RET proto-oncogene: paradigm for molecular medicine / Lois M. Mulligan, Charis Eng
  • Sect. 4. DNA repair and cancer
  • DNA repair pathways and changes in cancer / Vilhelm A. Bohr
  • Mutagenesis and tumor development in DNA mismatch repair-deficient mice / Susan E. Andrew, Peter M. Glazer, Frank R. Jirik
  • LOH and mitotic recombination / Maria Jasin
  • Sect. 5. Inherited mutations contributing to oncogenesis
  • Cancer susceptibility genes: germline mutations and polymorphisms conferring high risks of neoplasia / William P. Bennett
  • ATM: at the crossroads of DNA damage response, cell-cycle control, genome stability, and cancer / Galit Rotman, Yosef Shiloh
  • Molecular basis of predisposition to colon cancer / Nickolas Papadopoulos
  • Inherited susceptibility to breast cancer in women: high-penetrance and low-penetrance genes / Timothy R. Rebbeck
  • Sect. 6. DNA methylation, imprinting, and cancer
  • DNA hypomethylation and cancer / Melanie Ehrlich
  • Epigenetics and loss of gene function in cancer / Stephen B. Baylin, James G. Herman
  • Hypermethylator phenotypes in aging and cancer / Jean-Pierre Issa
  • 5-methylcytosine and human cancer mutations: lessons from p53 / Mikhail Denissenko, Gerd P. Pfeifer
  • Genomic imprinting and human neoplasia / Benjamin Tycko
  • DNA methylation and methyltransferases / En Li, Rudolf Jaenisch
  • Sect. 7. The contribution of karyotypic instability to cancer formation and tumor progression
  • Chromosome and karyotype instability to cancer formation and tumor progression
  • Chromosome and karyotype instability in human cancers and cancer-predisposing syndromes / Bernard Dutrillaux
  • Clonal evolution of neoplastic cell populations: lessons from solid tumor cytogenetics / Sverre Heim, Manuel R. Teixeira
  • The role of fragile sites in gene amplification and genome remodeling / Michelle Debatisse, Franck Toledo, Arnaud Coquelle
  • p53 and the development of genomic instability: possible role of topoisomerase I interaction / Amador Albor, Molly Kulesz-Martin
  • Sect. 8. New strategies for cancer therapies
  • Telomerase as a potential target for anticancer therapies / John K. Cowell
  • New p53-based strategies for cancer therapy / Ute M. Moll
  • Therapeutic resistance of erbB-2-overexpressing cancers and strategies to overcome this resistance / Dihua Yu, Mien-Chie Hung
  • The DNA repair protein O⁶-alkylguanine-DNA alkyltransferase as a target for cancer chemotherapy / Anthony E. Pegg, Meng Xu-Welliver, Natalia A. Loktionova
  • Acquired drug resistance driven by tumor cell genetic instability: circumvention by direct-acting anti-angiogenic vascular targeting agents / Robert S. Kerbel.