Physiology and pathophysiology of the islets of Langerhans /

Proceedings of the November 1994 meeting. The 59 contributions cover glucose recognition, the role for K (ATP) channels, intracellular messengers, modulation of islet release, beta-cell metabolism and Ca2+, beta-cell heterogeneity: building a homogeneous response from a heterogeneous population, ISS...

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Bibliographic Details
Corporate Authors: International Meeting of the Pancreatic Islet Study Group Alicante, Spain, SpringerLink (Online service)
Other Authors: Soria, Bernat (Editor)
Format: Conference Proceeding eBook
Language:English
Published: New York : Springer Science+Business Media, LLC, [1997]
Series:Advances in experimental medicine and biology ; v. 426.
Subjects:
Online Access:Connect to the full text of this electronic book
Table of Contents:
  • 1. Why Are Islets So Important? / Bernat Soria
  • 2. Hexose Recognition by the Pancreatic [beta]-Cell and the Gustatory Cell / A. Niki and H. Niki
  • 3. Glucose-Induced Time-Dependent Potentiation and "Run Down" of Insulin Secretion in Islets of Young Rats / Cheryl R. Bliss and Geoffrey W. G. Sharp
  • 4. ATP-Sensitive K[superscript +] Channel Closure Is Not an Obligatory Step for Glucose-Induced Priming of Pancreatic B-Cell / N. Taguchi, T. Aizawa and K. Hashizume
  • 5. Bursting Electrical Activity Generated in the Presence of K[subscript ATP] Channel Blockers: Pharmacology, Sensitivity to Intracellular pH and Modulation by Glucose Metabolism / Rosa M. Santos, Rui M. Barbosa and Celia M. Antunes [et al.]