Physiology and pathophysiology of the islets of Langerhans /
Proceedings of the November 1994 meeting. The 59 contributions cover glucose recognition, the role for K (ATP) channels, intracellular messengers, modulation of islet release, beta-cell metabolism and Ca2+, beta-cell heterogeneity: building a homogeneous response from a heterogeneous population, ISS...
| Corporate Authors: | , |
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| Other Authors: | |
| Format: | Conference Proceeding eBook |
| Language: | English |
| Published: |
New York :
Springer Science+Business Media, LLC,
[1997]
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| Series: | Advances in experimental medicine and biology ;
v. 426. |
| Subjects: | |
| Online Access: | Connect to the full text of this electronic book |
Table of Contents:
- 1. Why Are Islets So Important? / Bernat Soria
- 2. Hexose Recognition by the Pancreatic [beta]-Cell and the Gustatory Cell / A. Niki and H. Niki
- 3. Glucose-Induced Time-Dependent Potentiation and "Run Down" of Insulin Secretion in Islets of Young Rats / Cheryl R. Bliss and Geoffrey W. G. Sharp
- 4. ATP-Sensitive K[superscript +] Channel Closure Is Not an Obligatory Step for Glucose-Induced Priming of Pancreatic B-Cell / N. Taguchi, T. Aizawa and K. Hashizume
- 5. Bursting Electrical Activity Generated in the Presence of K[subscript ATP] Channel Blockers: Pharmacology, Sensitivity to Intracellular pH and Modulation by Glucose Metabolism / Rosa M. Santos, Rui M. Barbosa and Celia M. Antunes [et al.]