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mods:
  schema location: http://www.loc.gov/mods/v3 http://www.loc.gov/standards/mods/v3/mods-3-2.xsd
  title info:
    non sort: The
    title: generality of negative affect-induced pain modulation
  name:
    name part: Rhudy, Jamie Lynn.
    role:
      role term: creator
  type of resource: text
  genre: bibliography
  origin info:
    place:
      place term: xx
    date issued: 2002
    issuance: monographic
  language:
    language term: eng
  physical description:
    form: print
    extent: x, 130 leaves : illustrations ; 28 cm.
  abstract: Five studies built upon prior work that suggests negative affect can modulate pain. In Experiments 1 and 2 (between-subjects design), pain was tested before and after one of three emotion-induction manipulations that elicited negative affect. To elicit highly arousing negative affect, the shock manipulation exposed participants to surprising electrical stimulations. To elicit moderately arousing negative affect, the anticipation manipulation verbally threatened participants with shock (without exposure). A neutral condition just tested pain. To measure pain, Experiment 1 used pain ratings to a constant intensity and duration heat source (hand ipsilateral to shocks), and Experiment 2 used finger withdrawal to heat (contralateral to shocks). In Experiments 3-5 (within-subjects design), a conditioned fear paradigm was used to test the bilateral influence of highly arousing negative affect on finger withdrawal to fast-ramped heat (Exp 3), pain ratings to constant heat (Exp 4), and finger withdrawal to slow-ramped heat (Exp 5). It was hypothesized that low to moderately arousing negative affect would elicit hyperalgesia, whereas highly arousing negative affect would elicit hypoalgesia. Intense, highly arousing negative affect induced by shock and conditioned fear caused pain inhibition in every experiment, across three pain indices. Surprisingly, the anticipation manipulation resulted in a mild hypoalgesia in Experiment 1 and no group-level change in Experiment 2. Exploratory correlational analyses indicated that these unexpected results stemmed from individual differences in the affective state of participants. These analyses suggested that affect intensity, and not arousal alone, correlated with the direction of pain modulation. Low negative affect intensity caused no change in pain, moderate negative affect intensity caused hyperalgesia, and high negative affect intensity caused hypoalgesia. Physiological arousal correlated with the strength of the hypoalgesia, but no…
  note: by Jamie Lynn Rhudy.
  note: Vita.
  note: "Major Subject: Psychology".
  note: Issued also on microfiche from University Microfilm Inc.
  note: Ph. D. Texas A&M University 2002.
  note: Includes bibliographical references (leaves 118-129).
  subject:
    topic: Major psychology
  identifier: 30-60882 UMI
  identifier: http://proxy.library.tamu.edu/login
  related item:
    location:
      url: http://proxy.library.tamu.edu/login
  record info:
    record content source: TXA
    record creation date: 030407
    record change date: 20190325103406.0
    record identifier: in00001734952