Impact Of Dorsolateral Periaqueductal Gray Lesions On Shock-Induced Hyperalgesia.

Prior exposure to shock lowers vocalization thresholds to heat and facilitates the acquisition of conditioned fear when training is conducted in a difference context. These observations have been taken as evidence that shock exposure increases the effective impact of subsequent aversive stimuli, a p...

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Bibliographic Details
Main Author: McLemore, Sherilyn
Corporate Author: Texas A & M University. University Undergraduate Research Fellow Program
Format: Thesis Book
Language:English
Published: [College Station, Texas] : Texas A&M University, 1998.
Subjects:
Online Access:Link to OAK Trust copy
Available on OAKTrust.
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Summary:Prior exposure to shock lowers vocalization thresholds to heat and facilitates the acquisition of conditioned fear when training is conducted in a difference context. These observations have been taken as evidence that shock exposure increases the effective impact of subsequent aversive stimuli, a phenomenon known as hyperalgesia. The present study explores whether this hyperalgesia depends on neurons within the dorsolateral periaqueductal gray (d1PAG). Experiment 1 showed that lesioning either the rostral or caudal diPAG prevented the shock-induced reduction in vocalization thresholds. Experiment 2 showed those lesioned subjects also failed to exhibit facilitated learning after shock exposure. Taken together, these results suggest that the diPAG play a critical role in the production of shock-induced hyperalgesia.
Item Description:"Major Subject: Psychology II".
Physical Description:1 online resource (27 pages).
Digitized from print version held at Pickle Center High Density Storage, barcode 24829659
Bibliography:Includes bibliographical references: leaves 23-26.