The role of lysosomes in the pathogenesis of copper-induced hepatotoxicity.

Bibliographic Details
Main Author: Helman, Rowland Gayman
Other Authors: Bridges, Charles H. (degree committee member.)
Format: Thesis Book
Language:English
Published: 1981.
Subjects:
Online Access:Link to ProQuest Copy
Link to OAKTrust copy

MARC

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099 |a 1981  |a Dissertation  |a H478 
100 1 |a Helman, Rowland Gayman. 
245 1 4 |a The role of lysosomes in the pathogenesis of copper-induced hepatotoxicity. 
264 1 |c 1981. 
300 |a x, 103 leaves :  |b illustrations ;  |c 29 cm 
336 |a text  |b txt  |2 rdacontent 
337 |a unmediated  |b n  |2 rdamedia 
338 |a volume  |b nc  |2 rdacarrier 
500 |a "Major subject: Veterinary Pathology." 
500 |a Typescript (photocopy). 
500 |a Vita. 
502 |b Ph. D. in Philosophy  |c Texas A & M University  |d 1981 
504 |a Includes bibliographical references (leaves 85-95). 
520 3 |a The effects of altered lysosomal function on the pathogenesis of copper-induced hepatotoxicity were studied in beige and conventional mice. Copper loading was accomplished through daily intraperitoneal injections of aqueous copper chloride (8 mg/kg) for 1, 2, and 4 weeks. Selected tissues for morphologic studies, determination of intracellular copper distributions, and measurements of lysosomal lipoperoxides were collected after 0, 7, 14, and 28 days of treatment. In an additional study, beige and conventional mice receiving intraperitoneal injections of copper for 10 days were treated subsequently with oral chloroquine (120 mg/kg), and lysosomal labilization was measured as percent unbound acid phosphatase. Hepatic lesions resulting from progressive copper loading developed more quickly and were more severe in beige mice than in conventional mice. The primary hepatocellular lesion was demonstrated by light and transmission electron microscopy to be accelerated autophagocytosis. Beige mice had consistently higher levels of copper in the nuclear (N) and cytosolic (Cy) fractions with lower levels in the heavy (HM) and light (LM) mitochondrial fractions than did conventional mice. Levels of total hepatic copper were similar in both groups of mice except at day 14, when beige mice had higher levels. ... 
650 0 |a Hepatitis.  |0 http://id.loc.gov/authorities/subjects/sh85060288 
650 0 |a Liver  |x Diseases.  |0 http://id.loc.gov/authorities/subjects/sh85077755 
650 0 |a Liver  |x Necrosis.  |0 http://id.loc.gov/authorities/subjects/sh85077762 
650 0 |a Lysosomes.  |0 http://id.loc.gov/authorities/subjects/sh85079197 
650 4 |a Veterinary Pathology. 
655 7 |a Academic theses.  |2 lcgft  |0 http://id.loc.gov/authorities/genreForms/gf2014026039 
700 1 |a Adams, L. G.,  |e degree supervisor.  |0 http://id.loc.gov/authorities/names/no2013140475 
700 1 |a Bridges, Charles H.,  |e degree committee member. 
710 2 |a Texas A & M University,  |e degree granting institution.  |0 http://id.loc.gov/authorities/names/n80125885 
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