Biophysical model of oxygen transport and metabolic regulation in the bluegill.

Bibliographic Details
Main Author: Bryan, James Darnall
Other Authors: Folse, L. Joseph (degree committee member.), Hendricks, Fred S. (degree committee member.), Wu, Hsini (degree committee member.)
Format: Thesis Book
Language:English
Published: 1987.
Subjects:
Online Access:Link to ProQuest copy
Link to OAKTrust copy

MARC

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040 |a TXA  |b eng  |c TXA  |d OCLCQ  |d UMI  |d OCLCO  |d TXA 
049 |a TXAM 
099 |a 1987  |a Dissertation  |a B915 
100 1 |a Bryan, James Darnall. 
245 1 0 |a Biophysical model of oxygen transport and metabolic regulation in the bluegill. 
264 1 |c 1987. 
300 |a xiv, 323 leaves :  |b illustrations ;  |c 29 cm 
336 |a text  |b txt  |2 rdacontent 
337 |a unmediated  |b n  |2 rdamedia 
338 |a volume  |b nc  |2 rdacarrier 
500 |a Typescript (photocopy). 
500 |a Vita. 
502 |b Ph. D. in Wildlife and Fisheries Sciences  |c Texas A & M University  |d 1987 
504 |a Includes bibliographical references (leaves 267-272). 
520 3 |a Consideration of biophysical mechanisms and fish anatomy leads to a plausible model of oxygen transport and metabolic regulation. The first of the model's five major components is a cardiovascular regulation submodel describing steady-state blood flow through a purely resistive vascular network. Blood flow is driven by non-pulsatile blood pressures generated by an aneural heart obeying Laplace's Law and Starling's Law of the Heart. In the second submodel, steady-state oxygen uptake is described as a counter-current, bulk-flow, mass-transport process. The third submodel implements tissue autoregulation of blood flow based on changes in the number of open capillaries in response to tissue levels of dissolved oxygen. The fourth component regulates tissues metabolic rate and performance based on oxygen availability within the tissue. The final component is an osmoregulation model describing water and solute flux between the fish and the medium. The submodels are combined in a computer program for simulating routine metabolism of bluegill (Lepomis macrochirus). Agreement between simulated and observed responses to progressive hypoxia supports the assumptions and mechanisms incorporated in the model. Simulation results indicate that the response of the oxygen-transport system to reduction in ambient dissolved oxygen is the exchange of branchial diffusive limitation for tissue diffusive limitation. Tissues respond to hypoxia by opening more capillaries, thereby enhancing diffusion gradients and exchange characteristics both of branchial and tissue exchangers. Oxygen-transport costs are reduced by enhanced diffusive characteristics and elevated by convective costs and oxygen-utilization inefficiency. The net cost of the oxygen-transport system is roughly equal to the cost required to elevate the ambient concentration of dissolved oxygen to that which would generate spontaneous diffusion of oxygen to the tissues at the metabolic rate. The oxygen-transport and metabolic-regulation model invokes only fundamental biophysical mechanisms. The model's realistic performance suggests that extrinsic hormonal or neural influences--while they may condition or enhance the control inherent in the oxygen-transport system--are not required to produce fishes' characteristics responses to progressive hypoxia at constant temperatures. 
650 0 |a Bluegill. 
650 0 |a Fishes  |x Physiology. 
650 0 |a Metabolism  |x Regulation  |x Mathematical models. 
650 0 |a Oxygen  |x Physiological transport  |x Mathematical models. 
650 4 |a Major wildlife and fisheries sciences. 
655 7 |a Academic theses  |2 lcgft 
700 1 |a Folse, L. Joseph,  |e degree committee member. 
700 1 |a Hendricks, Fred S.,  |e degree committee member. 
700 1 |a Neill, William H.,  |e degree supervisor. 
700 1 |a Wu, Hsini,  |e degree committee member. 
710 2 |a Texas A & M University,  |e degree granting institution. 
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